Ana Tapia Abellán-rekin lankidetzan egindako argitalpenak (13)

2024

  1. Pathogenic NLRP3 mutants form constitutively active inflammasomes resulting in immune-metabolic limitation of IL-1β production

    Nature Communications, Vol. 15, Núm. 1

2023

  1. ASC oligomer favors caspase-1CARD domain recruitment after intracellular potassium efflux

    The Journal of cell biology, Vol. 222, Núm. 8

  2. Comment on: Effective ex vivo inhibition of cryopyrin-associated periodic syndrome (CAPS)-associated mutant NLRP3 inflammasome by MCC950/CRID3: Reply

    Rheumatology (Oxford, England), Vol. 62, Núm. 5, pp. e168-e169

  3. Measuring NLR Oligomerization III: Detection of NLRP3 and NLRC4 Complex by Bioluminescence Resonance Energy Transfer

    Methods in molecular biology (Clifton, N.J.), Vol. 2696, pp. 93-103

2022

  1. Effective ex vivo inhibition of cryopyrin-associated periodic syndrome (CAPS)-associated mutant NLRP3 inflammasome by MCC950/CRID3

    Rheumatology (United Kingdom)

2021

  1. Erratum: Addendum: MCC950 closes the active conformation of NLRP3 to an inactive state (Nature chemical biology (2019) 15 6 (560-564))

    Nature chemical biology

  2. Sensing low intracellular potassium by NLRP3 results in a stable open structure that promotes inflammasome activation

    Science Advances, Vol. 7, Núm. 38

2020

  1. Author Correction: Early endosome autoantigen 1 regulates IL-1β release upon caspase-1 activation independently of gasdermin D membrane permeabilization (Scientific Reports, (2019), 9, 1, (5788), 10.1038/s41598-019-42298-4)

    Scientific Reports

  2. Isolation of functional mature peritoneal macrophages from healthy humans

    Immunology and Cell Biology, Vol. 98, Núm. 2, pp. 114-126

2019

  1. Early endosome autoantigen 1 regulates IL-1β release upon caspase-1 activation independently of gasdermin D membrane permeabilization

    Scientific Reports, Vol. 9, Núm. 1

  2. MCC950 closes the active conformation of NLRP3 to an inactive state

    Nature Chemical Biology, Vol. 15, Núm. 6, pp. 560-564

2018

  1. NLRP3 lacking the leucine-rich repeat domain can be fully activated via the canonical inflammasome pathway

    Nature Communications, Vol. 9, Núm. 1

2017

  1. Development of an Acrylate Derivative Targeting the NLRP3 Inflammasome for the Treatment of Inflammatory Bowel Disease

    Journal of Medicinal Chemistry, Vol. 60, Núm. 9, pp. 3656-3671