Publicaciones (38) Publicaciones de Fernando Soler Pardo

2023

  1. Silencing of microRNA-106b-5p prevents doxorubicin-mediated cardiotoxicity through modulation of the PR55α/YY1/sST2 signaling axis

    Molecular Therapy - Nucleic Acids, Vol. 32, pp. 704-720

2022

  1. Critical warm ischemia time point for cardiac donation after circulatory death

    American Journal of Transplantation, Vol. 22, Núm. 5, pp. 1321-1328

2021

  1. The miRNA199a/SIRT1/P300/Yy1/sST2 signaling axis regulates adverse cardiac remodeling following MI

    Scientific Reports, Vol. 11, Núm. 1

2017

  1. Doxorubicin-induced oxidative stress: The protective effect of nicorandil on HL-1 cardiomyocytes

    PLoS ONE, Vol. 12, Núm. 2

2016

  1. Early oxidative damage induced by doxorubicin: Source of production, protection by GKT137831 and effect on Ca2+ transporters in HL-1 cardiomyocytes

    Archives of Biochemistry and Biophysics, Vol. 594, pp. 26-36

2015

  1. PDE2 activity differs in right and left rat ventricular myocardium and differentially regulates β2 adrenoceptor-mediated effects

    Experimental Biology and Medicine, Vol. 240, Núm. 9, pp. 1205-1213

  2. Single inhibition of either PDE3 or PDE4 unmasks β2-adrenoceptor-mediated inotropic and lusitropic effects in the left but not right ventricular myocardium of rat

    European Journal of Pharmacology, Vol. 765, pp. 429-436

2014

  1. Inhibition mechanism of the intracellular transporter Ca2+-pump from sarco-endoplasmic reticulum by the antitumor agent dimethyl-celecoxib

    PLoS ONE, Vol. 9, Núm. 7

2012

  1. Inhibition of the intracellular Ca2+ transporter SERCA (Sarco-endoplasmic Reticulum Ca2+-ATPase) by the natural polyphenol epigallocatechin-3-gallate

    Journal of Bioenergetics and Biomembranes, Vol. 44, Núm. 5, pp. 597-605

2011

  1. Passive Ca2+ overload in H9c2 cardiac myoblasts: Assessment of cellular damage and cytosolic Ca2+ transients

    Archives of Biochemistry and Biophysics, Vol. 512, Núm. 2, pp. 175-182

2009

  1. Mitochondrial damage as death inducer in heart-derived H9c2 cells: More than one way for an early demise

    Journal of Bioenergetics and Biomembranes, Vol. 41, Núm. 4, pp. 369-377

2008

  1. Characterization of the palytoxin effect on Ca2+-ATPase from sarcoplasmic reticulum (SERCA)

    Archives of Biochemistry and Biophysics, Vol. 478, Núm. 1, pp. 36-42

2007

  1. Cellular death linked to irreversible stress in the sarcoplasmic reticulum: The effect of inhibiting Ca2+-ATPase or protein glycosylation in the myocardiac cell model H9c2

    Archives of Biochemistry and Biophysics, Vol. 466, Núm. 2, pp. 194-202

2006

  1. Cytoplasmic Ca2+ signals and cellular death by apoptosis in myocardiac H9c2 cells

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1763, Núm. 9, pp. 937-947

2005

  1. Intracellular Ca2+ pools and fluxes in cardiac muscle-derived H9c2 cells

    Journal of Bioenergetics and Biomembranes, Vol. 37, Núm. 4, pp. 249-259

2004

  1. Functional approach to the catalytic site of the sarcoplasmic reticulum Ca 2+-ATPase: Binding and hydrolysis of ATP in the absence of Ca 2+

    Journal of Bioenergetics and Biomembranes, Vol. 36, Núm. 3, pp. 265-273

  2. Functional effect of hydrogen peroxide on the sarcoplasmic reticulum membrane: Uncoupling and irreversible inhibition of the Ca 2+-ATPase protein

    Archives of Biochemistry and Biophysics, Vol. 431, Núm. 2, pp. 245-251

2002

  1. Dissecting the hydrolytic activities of sarcoplasmic reticulum ATPase in the presence of acetyl phosphate

    Journal of Biological Chemistry, Vol. 277, Núm. 41, pp. 38127-38132

  2. Inhibition of sarcoplasmic reticulum Ca2+-ATPase by miconazole

    American Journal of Physiology - Cell Physiology, Vol. 283, Núm. 1 52-1

2001

  1. Testing the Versatility of the Sarcoplasmic Reticulum Ca 2+-ATPase Reaction Cycle When p-Nitrophenyl Phosphate Is the Substrate

    Journal of Biological Chemistry, Vol. 276, Núm. 11, pp. 7998-8004