Publicaciones en colaboración con investigadores/as de Universidad de Castilla-La Mancha (18)

2015

  1. Defects in subventricular zone pigmented epithelium-derived factor niche signaling in the senescence-accelerated mouse prone-8

    FASEB Journal, Vol. 29, Núm. 4, pp. 1480-1492

  2. Thalamus: Anatomy

    Brain Mapping: An Encyclopedic Reference (Elsevier Inc.), pp. 229-242

2012

  1. CCL2-Expressing astrocytes mediate the extravasation of T lymphocytes in the brain. Evidence from patients with glioma and experimental models in vivo

    PLoS ONE, Vol. 7, Núm. 2

2010

  1. Cholinesterase activity in brain of senescence-accelerated-resistant mouse SAMR1 and its variation in brain of senescence-accelerated-prone mouse SAMP8

    Journal of Neuroscience Research, Vol. 88, Núm. 1, pp. 155-166

  2. Cylindroma of the external auditory canal: Histopathology diagnostic

    Otology and Neurotology, Vol. 31, Núm. 3, pp. 546-547

  3. Mitochondria and calcium flux as targets of neuroprotection caused by minocycline in cerebellar granule cells

    Biochemical Pharmacology, Vol. 79, Núm. 2, pp. 239-250

2008

  1. 6-Hydroxydopamine activates the mitochondrial apoptosis pathway through p38 MAPK-mediated, p53-independent activation of Bax and PUMA

    Journal of Neurochemistry, Vol. 104, Núm. 6, pp. 1599-1612

  2. BAX and BAK proteins are required for cyclin-dependent kinase inhibitory drugs to cause apoptosis

    Molecular Cancer Therapeutics, Vol. 7, Núm. 12, pp. 3800-3806

  3. Farmacología de la neuroprotección en el ictus isquémico agudo

    Revista de Neurologia, Vol. 47, Núm. 5, pp. 253-260

  4. Is good old minocycline a new neuroprotective drug?

    Revista de Neurologia, Vol. 47, Núm. 1, pp. 31-38

  5. The level of butyrylcholinesterase activity increases and the content of the mRNA remains unaffected in brain of senescence-accelerated mouse SAMP8

    Chemico-Biological Interactions, Vol. 175, Núm. 1-3, pp. 332-335

2007

  1. Minocycline and cytoprotection: Shedding new light on a shadowy controversy

    Current Drug Delivery, Vol. 4, Núm. 3, pp. 225-231

  2. Reactive oxygen species and p38 mitogen-activated protein kinase activate bax to induce mitochondrial cytochrome c release and apoptosis in response to malonate

    Molecular Pharmacology, Vol. 71, Núm. 3, pp. 736-743

2006

  1. Pyruvate protects cerebellar granular cells from 6-hydroxydopamine-induced cytotoxicity by activating the Akt signaling pathway and increasing glutathione peroxidase expression

    Neurobiology of Disease, Vol. 24, Núm. 2, pp. 296-307

2005

  1. Activation of p53 and the pro-apoptotic p53 target gene PUMA during depolarization-induced apoptosis of chromaffin cells

    Experimental Neurology, Vol. 196, Núm. 1, pp. 96-103

  2. Involvement of mitochondrial potential and calcium buffering capacity in minocycline cytoprotective actions

    Neuroscience, Vol. 133, Núm. 4, pp. 959-967

  3. Malonate induces cell death via mitochondrial potential collapse and delayed swelling through an ROS-dependent pathway

    British Journal of Pharmacology, Vol. 144, Núm. 4, pp. 528-537

  4. Minocycline fails to protect cerebellar granular cell cultures against malonate-induced cell death

    Neurobiology of Disease, Vol. 20, Núm. 2, pp. 384-391